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Squalene secreted by tumors can modulate the NF-B pathway through p65, represses CXCL1 transcription, and thereby reduces the recruitment of MDSCs and TAMs into the tumor microenvironment (337)

ARA-290 operates through three complementary mechanisms identified across pre-clinical and clinical research: 1

Depending on the substrate in question, these steps can be performed with or without participation of a carrier lipid and, in some cases, are stimulated by the bacterial second messenger bis-(35)-cyclic dimeric guanosine monophosphate (c-di-GMP 49 )

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