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To assess the abilities of SOD1 WT and SOD1 G93A in generating lipid ROS, after transfection with SOD1 WT or SOD1 G93A construct, cells were loaded with BODIPY581/591 C11 and analyzed by flow cytometry

This review discusses metabolic signaling pathways pertaining to cysteine disposition in the brain during normal and pathological conditions highlighting recent findings on cysteine metabolism during aging and in neurodegenerative conditions such as Huntingtons disease and molybdenum cofactor deficiency among others

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