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Furthermore, its versatility makes it useful for a wide range of basic research models

Moreover, several NSCLC lines exhibited a significant elevation in maximal respiratory capacity that could not be explained by an increase in mitochondrial density (Fig

While early literature suggested that p53-dependent tumor suppression involved the induction of cell cycle arrest, senescence or apoptosis, the recent analysis of several transactivation-defective p53 mutants has called this model into question and it now appears that alterations in intracellular metabolism may account for the ability of p53 to suppress tumor formation [6870]

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