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In renal tissue, acute or chronic systemic injections of trimetazidine have been shown to reduce thiobarbituric acid reactive substances in immunosuppressant-induced renal dysfunction [178] and prevent SOD, glutathione peroxidase, catalase or glutathione decrease in ischemia/reperfusion injury in rats [179, 180]

Its a small detail that can have big downstream consequences

J Physiol 184, 239254

Specifically, the accumulation of misfolded CFTR in the endoplasmic reticulum (ER), as in the case of the F508del mutation, induces ER stress that fuels ROS production