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Under most physiological conditions, GSH buffers oxidants in the cytoplasm and mitochondria and supports nuclear repair processes, thereby increasing the threshold for regulated cell death and, in certain pathological settings, contributing to therapy resistance

Optimization of Dietary Folate or Low-Dose Folic Acid Supplements Lower Homocysteine but Do Not Enhance Endothelial Function in Healthy Adults, Irrespective of the Methylenetetrahydrofolate Reductase (C677T) Genotype. Journal of the American College of Cardiology, vol