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Nearly everything known comes from animal studies, so the human picture is still thin

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In agreement with these findings, we found that NQO1 overexpression under HFD favors an increase in the activities of CRAT and SIRT3 that buffers mitochondrial acetyl group availability while counteracting mitochondrial protein hyperacetylation in the muscle concomitantly with enhancement of glucose and NAD metabolism in liver, skeletal muscle and adipose tissues

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