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Starvation of cystine, a substrate used to synthesize GSH to prevent ferroptosis, was able to induce cell necroptosis and ferroptosis in TNBC via the GCN2-eIF2-ATF4-CHAC1 pathway [79]

The MRP proteins mediate not only GSH efflux, but they also export oxidized glutathione derivatives (e.g., glutathione disulfide (GSSG), S-nitrosoglutathione (GS-NO) and glutathione-metal complexes), as well as other glutathione S-conjugates [18]

Scientific evidence has shown the important immune-inflammatory roles of glutamine availability and/or glutamine metabolism in stressful and/or catabolic situations

Patients who are pregnant or breastfeeding, who have known sulphur or sulphite sensitivity, or who have certain medical conditions may need to defer or avoid treatment