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Ishihara Y, Tsuji M, Kawamoto T, Yamazaki T (2016) Involvement of reactive oxygen species derived from mitochondria in neuronal injury elicited by methylmercury

While the regulation of MTHFR activity is closely controlled by SAM at C-terminal regulatory domain, more studies indicated that the human MTHFR enzyme activity is also regulated by multiple phosphorylated sites on a serine-rich N-terminal extension region 20

Crossovers (COs) between homologues create the physical connections that enable their biorientation on the meiotic spindle 1 , yet the very same DNA exchanges are suppressed in somatic cells because they can provoke deleterious chromosomal rearrangements and loss-of-heterozygosity 2