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These two papers are the load-bearing bpc-157 in vitro studies for the VEGFR2/eNOS hypothesis

Proposed mechanisms of AOD-9604: Stimulates lipolysis (fat breakdown) Increases fat oxidation Promotes mobilization of stored fat Influences lipid metabolism Importantly, AOD-9604 does not appear to significantly increase growth hormone levels, IGF-1 levels, water retention, or organ growth

As per your suggestion of lowering the pressure, there are some bacteria that can survive in a vacuum (BBC, 2010) so you might only be able to kill the bacteria by raising the pressure