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Nitrosative stress and redox-cycling agents synergize to cause mitochondrial dysfunction and cell death in endothelial cells

Therefore, pharmacological agents that inhibit NF-B or p38 MAPK represent a promising class of drugs for repurposing in tinnitus, particularly for prevention in high-risk scenarios or early intervention in acute cases, by broadly suppressing the inflammatory milieu that facilitates the establishment of tinnitus see in Figure 2 , and

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