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Description

Biomedicines 10, 3285

Under different ferroptosis induction conditions, p53 promotes ferroptosis partly by inhibiting SLC7A11 expression, while p53 deficiency promotes ferroptosis resistance by SLC7A11 upregulation

Animal models have provided insights into the biology of prolactin proteins and offer compelling evidence that the different prolactin isoforms each have independent biological functions

Further studies are needed to determine how the glutathionylation of RBP proteins occurs during the progression of ALS and how it is linked to the pathogenesis of ALS