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Dysfunction of key astrocytic molecular systems, such as those mediated by the EAAT2, AQP4, and purinergic receptors, contributes to neurodegenerative and neurological diseases, including AD, ALS, epilepsy, MS, and sleep fragmentationassociated pathology

Interestingly, the same pathway may also be utilized during inflammatory responses, mediated by the tumor necrosis factor-alpha (TNF-)

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In comparison to the wild type mice, APP/PS1 mice experienced significant synaptic loss as well as a reduction in the number of neuronal cells in the cerebral cortex