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Emerging studies have reported that increased anti-dsDNA autoantibodies or dsDNA immune complexes strongly promote platelet activation and thrombosis events by via binding to the FcRIIA ( via platelet activation through FcRIIA stimulation induces a circulatory autoantigenic load that ultimately leads to SLE pathogenesis ( Mitochondria and neuropsychiatric SLE Neuropsychiatric (NP) involvement and cognitive symptoms are some of the most severe manifestations of SLE

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This partially suppressed VEGFA and IL8 expression, suggesting that STAT6 contributes, at least in part, to ferroptosis-mediated VEGFA and IL8 production (Fig