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Their distribution in ion channels embedded in membranes may be a relevant factor, as they can regulate these either directly via their physical location in membranes or indirectly as second messengers, which control innumerable aspects of cell physiology

Furthermore, oxidative stress contributes to neuronal damage and mediates neuroinflammation, blood-brain barrier (BBB) disruption, and the production of spasminogen following SAH ( CU has demonstrated multiple significant neuroprotective effects, particularly in conditions such as ischemic stroke, traumatic brain injury, and intracranial hemorrhage (Thiyagarajan and Sharma, 2004)

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