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The relationship between this response and both glutathione homeostasis and overall antioxidant capacity is not yet clear, and additional research is planned to investigate this further

The data we obtained indicate that the reason for the decrease in the sensitivity of cells with a gmhA deletion to antibiotics is the depletion of the pool of low-molecular-weight thiols, as is observed in violation of the metabolism of cysteine and glutathione, while an increase in the content of cystine, the oxidized form of cysteine, or cysteine leads to an increase in the sensitivity of these cells to antibiotics

NPs offer the possibility of personalized therapies, but this requires a deeper understanding of disease mechanisms and patient-specific responses to treatment

Supports intracellular redox balance Promotes detoxification pathways Contributes to immune system regulation Reinforces oxidative stress defense References 1