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Increased biliary glutathione disulfide release in chronically ethanol-treated rats

Clinical Microbiology Reviews

HOCl is an oxidative substance produced in vivo by neutrophil myeloperoxidase at the site of inflammation and can cause damage to proteins including enzymes

Although this phenomenon is considered the primary pathway of melanocyte destruction in vitiligo, the presence of melanocyte-specific T cells is not sufficient, as they have also been found in healthy individuals, showing an anergic phenotype.19,20 Regulatory T cells The imbalance between pro- and anti-inflammatory signaling is crucial in the pathogenesis of vitiligo