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This is possible due to cysteine residues present on KEAP1 that can be oxidized and prevent the otherwise inevitable ubiquitination and degradation of NRF2

In individuals with primary coenzyme Q 10 deficiency, early treatment with high-dose coenzyme Q 10 supplementation (1030 mg/kg/day in children and 1.23.0 g/day in adults) may improve the pathological phenotype, yet the effectiveness depends on the type of mutations affecting the coenzyme Q 10 biosynthetic pathway (1, 26)

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Thus, our attention instead focused on proteins and BSA was a likely candidate