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The MIF homologue D-dopachrome tautomerase promotes COX-2 expression through beta-catenin-dependent and -independent mechanisms

Another possibility is that dietary restriction may indeed induce the expression of neuroprotective proteins in motor neurons, such as growth factors and stress proteins, but the pathogenic pathways involved in Cu/Zn SOD mutations are not affected by dietary restriction

Elevated PHGDH restricts serine biosynthesis, while ASS1 knockdown partially rescues TNBC progression under serine deprivation [48]

From reading the papers cited, you will find that these studies did not use native fragment 176-191