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The principal mechanisms that link lipid metabolism with neurodegeneration in AD include neuronal signaling pathways, blood-brain barrier (BBB) disruption, mitochondrial dysfunction, OS, and inflammation, which ultimately lead to synaptic loss and memory impairment (279, 280)

Specifically, IL-22 upregulates lncRNA-UCL, enhances claudin-1 expression by sequestering miR-568, reduces pro-inflammatory cytokines (TNF-, IL-1, IL-6, IL-17A), and thus promotes IEC proliferation while preventing apoptosis [84]

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Increased Energy Levels Supplementing with sermorelin may have a dual benefit: One is boosting muscle mass, and the other is facilitating fat loss