glutathione and triple negative breast cancer CD36 enhances sensitivity of cells to palmitate-induced ferroptosis cd44 glutathione Receptor-Mediated Ferroptosis Induction
cd44 glutathione Receptor Mediated Ferroptosis Induction by Hyaluronic Acid Carbon Quantum Dots in Triple Negative Breast Cancer Cells Through Downregulation of SLC7A11 Pathway Reduction sensitive CD44 receptor targeted hyaluronic acid Excessive glutathione intake contributes to chemotherapy resistance in breast cancer: a propensity score matching analysis PMC Heterogeneity and transcriptional drivers of triple negative breast cancer: Cell Reports Activation of the eIF2 ATF4 axis drives triple negative breast cancer radioresistance by promoting glutathione biosynthesis ScienceDirect
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