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Quantitative analyses reveal that both cytokine concentration and temporal expression patterns significantly influence subsequent neurodegenerative progression, with chronic low-level inflammation potentially exerting more detrimental effects than acute inflammatory responses due to the cumulative impact of sustained neuronal exposure to inflammatory mediators (36, 37)

The results showed that BRG1 could bind to the TSS of GCLC, and the peak was overlapped with that of anti-ACTL6A ChIP-seq (Supplementary Fig

10.1385/BTER:87:1-3:133 Pubmed Abstract CrossRef Google Scholar View reference in article 35

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