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203 Targeting PRMT7 reduces glycine decarboxylase expression, leading to the reprogramming of glycine metabolism and production of methylglyoxal, impairing the self-renewal capacity of leukemia stem cells (LSCs) and slowing the progression of leukemia

In vitro , the activated HGF binding to the c-Met drives the phosphorylation of c-Met tyrosine residues, then Wnt-independent nuclear translocation of -catenin is induced by HGF (Monga et al., 2002)

BPC-157 excels at localized tissue repair and concentrates at injection sites, while TB-500 distributes systemically due to its lower molecular weight and longer half-life

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