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A recent clinical trial examining the hepcidin mimetic, Rusfertide (PTG-300), for use in hereditary hemochromatosis demonstrated reduction in both transferrin saturation and serum iron during treatment, reduced requirement for phlebotomy during the study period, and resulted in control of hepatic iron concentration [163]

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Collectively, these studies underscore the pivotal role of -syn in PD-related ferroptosis and neurodegeneration, providing a rationale for therapeutic strategies targeting ferroptosis

Another mechanism of ER stress associated with -syn is the inhibition of protein trafficking to the Golgi apparatus, which interferes with protein maturation and induces stress [119, 120]