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doi: 10.1007/s00422-010-0372-z

In the late 1980s, the first murine animal model studies demonstrated that the cytokine cascade could alter bone marrow responses during sepsis 16 , leading to permanent modifications in the gene expression of hematopoietic stem and progenitor cells (HSPCs) 17

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Mitochondrial dysfunction not only impairs energy metabolism but also promotes further A accumulation and tau pathology, establishing a harmful feedback loop (Castellani et al., 2002