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Given these results, we measured the ability of a larger set of Aur R lines to compete with wild-type YFP-expressing RH strain parasites in the absence or presence of auranofin by quantifying their contribution to the culture at 6 days (Figure 2)

Genome-wide association studies (GWAS) identify PICALM as a major AD risk factor, second only to APOE and BIN1 (Van Acker et al., 2019)

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In a nonalcoholic steatohepatitis (NASH) model, human UCMSCs derived Exos significantly alleviated hepatocellular steatosis and suppressed inflammatory responses and oxidative stress by activating the Nrf2/NQO-1 pathway [137]