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In gastric cancer, STAT3 regulates ferroptosis primarily through its own histone acetylation, which increases its gene-level expression and promotes the expression of downstream direct targets GPX4, SLC7A11, and FTH1, thereby exerting an anti-ferroptotic effect [231]

Recent developments in biomedical science emphasize the involvement of free radicals in many diseases, such as brain dysfunction, cancer, heart disease, and immune system [6]

The availability of nutrients in the circulation might also have a direct effect on the production of IGF-I

The literature also reports that they are essential substances involved in the proper function and development of nerve cells (86)