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This patient's normal vital signs and isolated joint pain without any signs of encephalopathy make a urea cycle defect unlikely

Int J Mol Sci Theophylline Attenuates BLM-Induced Pulmonary Fibrosis by Inhibiting Th17 Differentiation

While copper may contribute to intracellular oxidative stress by participating in the Fenton and Haber-Weiss reactions or by interfering with iron homeostasis, either directly or indirectly through the formation of hydroxyl radicals, experimental evidence linking copper deficiency and the formation of SOD1 aggregates to the progression of PD is also discussed in terms of its therapeutic implications (119,120)