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Cultured human keratinocytes have been suggested as good models to predict dermal irritancy of substances in human subjects 31

Tendon injuries initially suffer from: Poor blood supply Limited vascularity Slow nutrient delivery In rodent models, BPC-157 appears to: Upregulate VEGFR2 signalling Increase capillary density Improve microvascular perfusion Mechanistically: BPC-157 VEGFR2 activation PI3K/Akt pathway eNOS phosphorylation Increased nitric oxide Improved angiogenesis More vascularity = better nutrient and oxygen delivery to fibroblasts

Depending on the individual, different results may be achieved

Differential expression of HSPA1 and HSPA2 proteins in human tissues