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Gene ontology analysis of proteins altered by BSO treatment (bottom)

Genetic spectrum and clinical early natural history of glucose-6-phosphate dehydrogenase deficiency in Mexican children detected through newborn screening

7d), confirming that the need to increase mitochondrial mass in 5FU-treated cells is driven by a dependence on increased mitochondrial activity, and metformin can block this mitochondrial adaptation to prevent drug tolerance in tumours

ACh exerts tonic effects to maintain constriction of the lower esophageal sphincter, with ACh signaling compromised in a rat model of gastrointestinal reflux disease (210)