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A study of iodothyronine 5-monodeiodinase activities in normal and pathological tissues in man and their comparison with activities in rat tissues

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Biochemically, it is becoming clear that ferroptosis is primarily triggered by the massive lipid peroxidation-mediated membrane rupture in an iron-dependent manner and modulated by lipid repair systems involving glutathione (GSH) and phospholipid peroxidase glutathione peroxidase 4 (GPX4) (Yang et al., 2014)

Thus, enhanced intracellular trafficking of fatty acids in the lipid laden liver of NAFLD patients may be shunting harmful fatty acids to storage, thereby promoting steatosis