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Therefore, understanding whether IF can mitigate these factors by improving both inflammatory and oxidative stress markers, as well as liver enzyme profiles, could offer a novel, non-pharmacological strategy for managing RA and its associated metabolic complications in this vulnerable population

Responsible for the hydrolysis of CysGly is the neuronal aminopeptidase N, as demonstrated by the ability of an inactivating specific antibody against aminopeptidase N to prevent the utilization of CysGly for neuronal GSH synthesis (Dringen et al

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