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Additionally, it has shown potential neuroprotective and cardioprotective activities in various experimental models, making it a widely explored peptide in tissue repair and regenerative medicine research

Forbes, J., & Krishnamurthy, K

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Paradoxically, DUSP16 knockdown in murine dendritic cells results in an elevation of antigen-presenting ability, leading to an increased proliferation of Th17 cells isolated from mice with the induction of experimental autoimmune uveitis [107]