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Its unique properties make it a valuable addition to an overall antioxidant strategy

105 Cardiac IGF1R overexpression induced physiological cardiac hypertrophy in mice, which was however blunted by cardiomyocyte-specific co-expression of a dominant negative PI3K(dnPI3K) p110 mutant

It is required for mitochondrial energy metabolism, normal nervous system function, red blood cell development and maturation, DNA synthesis and the methylation cycle, and has antioxidant effects at the cellular level