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Metformins anti-inflammatory effects, mediated through AMPK activation, help to suppress NF-B signaling and reduce the production of inflammatory mediators, thereby potentially mitigating these harmful lipid byproducts and their neurotoxic consequences [201,202]

This work has not yet clarified the priority and interaction mechanisms of ferroptosis within this network, limiting precise interpretation of its role across different injury phasesacute inflammatory response, subacute repair, and chronic glial scar formation

A metabolic modeling approach reveals promising therapeutic targets and antiviral drugs to combat COVID-19

Microbiome stability in healthy populations obviates frequent re-profiling