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More globally, the development of phMRI and other molecular imaging biomarkers can reveal therapeutic mechanisms, which will allow drug development and treatment to be tailored toward specific neural circuits and patient populations

[5] Systemically elevated estradiol exerts negative feedback on the hypothalamic-pituitary-gonadal axis, reducing secretion of luteinizing hormone and follicle-stimulating hormone, a property leveraged therapeutically in certain hypoestrogenic states to stabilize hormonal milieu and mitigate vasomotor volatility

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Since plasma levels of IGF-I are important mediators of most of the peripheral actions of GH, it is reasonable to assume that there must be important relationships between IGF-I and gastric ghrelin secretion