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Acute mitochondrial GPX4 inactivation upregulates DHODH metabolic flux, accelerating CoQ10H 2 synthesis to neutralize lipid radicals, compensate for GPX4 deficiency and inhibit mitochondrial inner membrane ferroptosis (109)

This type of damage is known as oxidative damage . As we learned in the UCP2 and SOD2 traits, ROS are produced during cell respiration the process by which cells derive chemical energy

Wenn durch den Lebensstil, Krankheiten, Umweltfaktoren oder das Alter oxidativer Stress zunimmt, bersteigt der Glutathionverbrauch die Glutathionsynthese

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