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This cascade leads to neuronal apoptosis and energy deficits, particularly in the prefrontal cortex and hippocampus ( 4.3 Excitotoxicity Synthetic cathinones disrupt glutamatergic signaling by enhancing glutamate release and impairing astrocytic uptake (Zhang and Bhavnani, 2006)

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However, for both teens and older menstruating women, when Depo is stopped, rising estrogen levels with menstrual cycle recovery and persistent progestin (acting like progesterone to cause increased new bone formation) together cause an increase in bone density

Pregnant, lactating females and patients with oral or topical treatment of melasma 1 month before the study were excluded from the study