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notably, mRNA levels of Tac2 (the mouse ortholog of human TAC3 ) were elevated, while those of Kiss1 receptor, Kiss1r , and estrogen receptor, Esr1 , were unaltered (Fig

[68] [69] Mechanisms of GPCR signal termination [edit] As mentioned above, G-proteins may terminate their own activation due to their intrinsic GTPGDP hydrolysis capability