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Beijing, PRC: Oxford University Press;Cornell University Press

Another recurring limitation emerges on antioxidant-based interventions in ASD, constrained by a fundamental methodological gap: most clinical studies report behavioral or functional improvements without directly measuring oxidative-stress biomarkers

Collectively, the above results indicate that the p38 MAPK/STAT6 signalling pathway contributes to ferroptosis-mediated VEGFA and IL8 expression

The interaction between aPL and targets like 2GPI triggers the activation of endothelial cells, platelets, monocytes, and neutrophils, as well as the complement and coagulation systems, forming a key molecular mechanism underlying APS pathogenesis (Knight and Erkan, 2024)