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Neurovascular unit dysfunction and neurodegenerative disorders

The free (unbound) T3 is what can enter and affect the body tissues

Interestingly, astrocytes (which protect neurons and promote sprouting) are activated because glial fibrillary acidic protein (GFAP) immunoreactivity increases in the striatum and indusium griseum upon METH treatment, thereby implying complex mechanisms involving guidance molecules and cytotoxic mediators in the neurotoxic consequences of METH ( FIGURE 4 Long-Term Neurotoxicity The effects of long-term exposure to METH are different from those of acute injury