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The calcineurin-TFEB-p62 pathway mediates the activation of cardiac macroautophagy by proteasomal malfunction

However, animal reproduction studies are not always predictive of human response

In hyperosmolar CECs, 1,5-diCQA prevented apoptosis and restored cell survival

Clinical studies have shown that coagulation activation during ECMO initially leads to the deposition of fibrin, platelets, and leukocytes on the circuit surface (63), which can develop into sarcoid thrombosis in some patients (64)