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Chemotherapy-related cardiomyopathy One of the major side effects of applying doxorubicin is potentially fatal cardiovascular toxicity, such as congestive HF and cardiomyopathy [145]

Additionally, a study found that in SCI animal models, the IncGm36569/miRNA-5627-5p/FSP1 axis was inhibited through molecular sponge action, thereby targeting this axis to inhibit neuronal ferroptosis (Shao et al., 2022)

However, M2 macrophages are also implicated in promoting fibrosis, potentially via TGF- production or, a topic of ongoing discussion, direct transdifferentiation into myofibroblasts [10]

Methods: Participants (pts) with uHCC and no prior ICI therapy received STRIDE (T 300 mg + D 1500 mg [1 dose during C1] then D 1500 mg once every 4 weeks [Q4W]), T75+D (T 75 mg + D 1500 mg Q4W [4 doses] then D 1500 mg Q4W), D (1500 mg Q4W), T (750 mg Q4W [7 doses] then Q12W), or D+B (D 1120 mg + B 15 mg/kg Q3W)