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The red blood cell is primarily involved in the maintenance of redox balance, with hemoglobin being a major factor in initiating oxidative stress within the erythrocyte (Rifkind and Nagababu, 2013)

Mechanistically, HGF inhibits inflammatory M1-like macrophage production of pro-inflammatory cytokines and chemokines (such as IL-1, IL-6, TNF, CXCL1, CXCL10, and CCL2) by disrupting nuclear factor NF-B signaling (Giannopoulou et al., 2008) through the enhancement of the heme oxygenase-1 (HO-1) transcriptional pathway and the inactivation of the GSK-3 pathway (Kamimoto et al., 2009a,b

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The inhibitory activity of CUR/miR155@DssD-Hb NPs revealed minimal variance from CUR@DssD-Hb NPs in both B16F10 and 4T1 cells