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Skin T cell inflammatory responses are hardwired in the thymus by oxysterol sensing via GPR183 and calibrated by dietary cholesterol

The pathogenesis of vitiligo may also be influenced by the emerging processes of regulated cell death known as ferroptosis, necroptosis and pyroptosis [25]

This is primarily mediated by the anti-apoptotic properties of sphingosine-1-phosphate (S1P) in HDL, which prevents the opening of mitochondrial permeability transition pores by activating the SAFE (Survivor Activating Factor Enhancement) and RISK (Reperfusion Injury Salvage Kinase) pathways

Moreover, by affecting the synthesis of this specific type of lipid, fatty acids can potentially regulate ETC activity in thus mitochondria, thus directly affecting cellular bioenergetics of tumor cells [66]