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Using a murine model of heart failure, Xu et al 157 demonstrated that the soluble epoxide hydrolase inhibitor 1-adamantan-3-(5-(2-(2-ethylethoxy)ethoxy)pentyl)urea could effectively enhance the biological activities of EETs and thus improve pressure overload- or Ang II (angiotensin II)-induced myocardial hypertrophy and ventricular arrhythmias

The mice then received 5%, 9%, or 12% water-content suppositories via the rectum

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Concretely, enhanced glycolysis in PCa cells leads to HIF-1 lactylation, upregulating KIAA1199, accompanied by increased secretion of VEGFA, ultimately advancing angiogenesis and vasculogenic mimicry