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Conversely, primary or secondary defects of the intestinal barrier can lead to excessive entrance of dietary or microbe-derived macromolecules, which are putative contributors to the pathogenesis of a spectrum of human diseases, including food allergy and inflammatory bowel diseases (IBDs), and could even be related to autoimmune diseases and metabolic syndrome

However, normoxic elevation and activation of HIF can be caused either by loss/inactivating mutations in VHL (occurring in most clear cell renal cell carcinomas) or by oncogenic pathways that increase the transcription, translation, and/or activity of the HIF- subunit in non-RCC tumors [6]

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