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223 ER can increase the proliferation of ectopic endometrial lesions and prevent apoptosis, thus promoting the progression of endometriosis

Entrance of the infected cells into a hypometabolic state was also shown by downregulation of glycolysisindependent transcriptional and translational pathways, although downregulation of these pathways did not reach the same level of convergence among the databases examined as compared to the glycolytic pathway (Appendix Table S1)

Interestingly, we found that succinate accumulated only in Gclc- deficient B cells and not in Gclc- deficient effector T cells nor Tregs 43,44