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In mouse models, GPER activation reduces inflammatory factor secretion and astrocyte activation following optic nerve injury by inhibiting nuclear factor kappa-light-chain enhancer of activated B cells (NF-B) signaling, protecting retinal neurons and alleviating neuroinflammation [71, 72]

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562 Third, ccf-mtDNA can be utilized for dynamic treatment response monitoring and individualized dose adjustments through machine learning models

Second, there is individual variability in SUD, requiring individualized treatment plans