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So, artificially prolonging half-life through the use of a sustained-release formulation is another strategy to reduce drug adverse effects

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Evidence was grouped by peptide agent and clinical domain, including tendon and ligament healing, cartilage and osteoarthritis, bone remodeling and fracture repair, wound healing, and postoperative recovery

NLRP3 itself and other inflammasome components are redox-regulated and share a sensitivity to oxidative stress [86]