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Feature · Product Review
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Description

The rationale undergoes the imbalance between oxidative stress and the antioxidant defense system

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Moderate HO-1 activation stabilizes plaques via antioxidative effects, whereas high HO-1 expression promotes macrophage ferroptosis, matrix metalloproteinase activity, and M0 macrophage infiltration, resulting in fibrous cap thinning and plaque instability (86)

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This review explores recent advances in nanoparticle (NP)-based strategies for HBV and HCV therapy, focusing on design principles, delivery platforms, and translational applications

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